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相关论文: Aging as a process of accumulation of Misrepairs

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It is now increasingly realized that the underlying mechanism which governs aging (ageing) is a complex interplay of genetic regulation and damage-accumulation. "Aging as a result of accumulation of 'faults' on cellular and molecular…

组织与器官 · 定量生物学 2011-03-31 Jicun Wang , Thomas Michelitsch , Arne Wunderlin , Ravi Mahadeva

Misrepair accumulation theory is a novel theory in interpreting aging phenomena. With a new concept of Misrepair, this theory reaches a unified understanding of aging: aging of an organism is a result of accumulation of Misrepairs of its…

组织与器官 · 定量生物学 2017-01-05 Jicun Wang-Michelitsch , Thomas M. Michelitsch

Aging changes including age spots and atherosclerotic plaques develop in an inhomogeneous and accelerated manner. For understanding this phenomenon, some aging changes are analyzed by Misrepair mechanism, a mechanism proposed in…

其他定量生物学 · 定量生物学 2018-02-07 Jicun Wang-Michelitsch , Thomas Michelitsch

In Misrepair-accumulation theory, we have proposed a Misrepair mechanism for interpreting aging. However, Misrepair mechanism is also important in biological adaptation. Misrepair is a strategy of repair for increasing the surviving chance…

组织与器官 · 定量生物学 2017-01-05 Jicun Wang-Michelitsch , Thomas M. Michelitsch

Tissue fibrosis is the phenomenon that a tissue has progressive deposition of collagen fibers with age. Tissue fibrosis is associated with aging of most of our organs, and it is the main pathology in arteriosclerosis, chronic…

组织与器官 · 定量生物学 2017-09-07 Jicun Wang-Michelitsch , Thomas M. Michelitsch

Atherosclerosis is a disease characterized by the development of atherosclerotic plaques (APs) in arterial endothelium. The APs in part of an arterial wall are inhomogeneous on size and on distribution. In order to understand this…

组织与器官 · 定量生物学 2017-09-07 Jicun Wang-Michelitsch , Thomas M. Michelitsch

The mortality rate of many complex multicellular organisms increase with age, which suggests that net aging damage is accumulative, despite remodeling processes. But how exactly do little mishaps in the cellular level accumulate and spread…

组织与器官 · 定量生物学 2017-12-12 Daniel Suma , Aylin Acun , Pinar Zorlutuna , Dervis Can Vural

Age spots are the brown spots that develop in the skin but change in color and shape with time. To understand the mechanism of development of age spots, characteristics of age spots are analyzed by Misrepair mechanism, a mechanism…

组织与器官 · 定量生物学 2018-02-06 Jicun Wang-Michelitsch , Thomas M Michelitsch

Although species longevity is subject to a diverse range of selective forces, the mortality curves of a wide variety of organisms are rather similar. We argue that aging and its universal characteristics may have evolved by means of a…

种群与进化 · 定量生物学 2013-02-01 Dervis Can Vural , Greg Morrison , L. Mahadevan

Many theories have been proposed to answer two questions on aging: "Why do we age?" and "How do we age?" Among them, evolutionary theories are proposed to interpret the evolutionary advantage of aging, and "saving resources for group…

其他定量生物学 · 定量生物学 2017-01-04 Jicun Wang-Michelitsch , Thomas Michelitsch

Aging remains a fundamental open problem in modern biology. Although there exist a number of theories on aging on the cellular scale, nearly nothing is known about how microscopic failures cascade to macroscopic failures of tissues, organs…

组织与器官 · 定量生物学 2017-07-10 Aylin Acun , Dervis Can Vural , Pinar Zorlutuna

Hutchinson-Gilford Progeria syndrome, Werner syndrome, and Cockayne syndrome are three genetic disorders, in which the children have premature aging features. To understand the phenomenon of premature aging, the similarity of aging features…

组织与器官 · 定量生物学 2017-09-07 Jicun Wang-Michelitsch , Thomas M. Michelitsch

We propose a new theory for aging based on dynamical systems and provide a data-driven computational method to quantify the changes at the cellular level. We use ergodic theory to decompose the dynamics of changes during aging and show that…

定量方法 · 定量生物学 2025-04-18 Farhan Khodaee , Rohola Zandie , Yufan Xia , Elazer R. Edelman

Based on the study of cellular aging using the single-cell model organism of budding yeast and corroborated by other studies, we propose the Emergent Aging Model (EAM). EAM hypothesizes that aging is an emergent property of complex…

定量方法 · 定量生物学 2024-07-09 Hong Qin

Development of a tumor is known to be a result of accumulation of DNA changes in somatic cells. However, the processes of how DNA changes are produced and how they accumulate in somatic cells are not clear. DNA changes include two types:…

组织与器官 · 定量生物学 2017-01-05 Jicun Wang-Michelitsch , Thomas M. Michelitsch

The question of why we age is a fundamental one. It is about who we are, and it also might have critical practical aspects as we try to find ways to age slower. Or to not age at all. Different reasons point at distinct strategies for the…

种群与进化 · 定量生物学 2020-11-10 André C. R. Martins

Aging is a universal consequence of life, yet researchers have identified no universal theme. This manuscript considers aging from the perspective of entropy, wherein things fall apart. We first examine biological information change as a…

种群与进化 · 定量生物学 2026-04-13 Stephan Baehr , Hans Baehr

A theory of aging based on the principles of the kinetics of chemical reactions and the rules of natural selection of organisms is proposed. The theory is based on a hypothesis that the biochemical processes in the organism can be described…

种群与进化 · 定量生物学 2024-01-09 Alexey Kondyurin

At any moment in time, evolution is faced with a formidable challenge: refining the already highly optimised design of biological species, a feat accomplished through all preceding generations. In such a scenario, the impact of random…

种群与进化 · 定量生物学 2024-02-20 Alessandro Fontana , Marios Kyriazis

The network concept is increasingly used for the description of complex systems. Here we summarize key aspects of the evolvability and robustness of the hierarchical network-set of macromolecules, cells, organisms, and ecosystems. Listing…

分子网络 · 定量生物学 2010-10-05 Huba J. M. Kiss , Agoston Mihalik , Tibor Nanasi , Balint Ory , Zoltan Spiro , Csaba Soti , Peter Csermely
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